The Rh Negative Blog

The History of Pandemics

This subject matter is of great interest to me for many reason.

A big one:

Who didn’t die and why?

Having examined infections of viral origin, there were plenty of data indicating and sometimes proving resistance based on both, ABO and Rh factor.

While in some cases ABO or Rh factor might be directly related to having resistance, there is also the possibility that data can show unusual frequencies based on individuals with certain blood types being more likely to carry other genes that can make them resistant or even immune.

Out of all the rh negatives included in the study, all tested negative for Chikungunya. Even the one where all rh positive family members were infected.

We are currently in the middle of the SARS-2 coronavirus pandemic where a recent study that wasn’t peer-reviewed suggested blood type O having some type of protection and blood type A being at high risk.

Is such a study credible?

First let us check if it is possible:

The answer is clearly “yes”.

Dismissing it would be foolish. Considering it possible, yet requesting more studies to confirm seems reasonable.

A researcher’s mind never rests.

A tired mind will always be behind.

People who flat-out dismiss are usually not up-to-date on research either.

Ebola has also shown interesting results.

The results indicate that persons with blood group A get infected less and survive more in the event of Ebola exposure. It appears that persons with blood group B get infected more and survive less in the event of Ebola exposure. The seemingly high death rate of infected with blood Group O is an artifact produced by the apparent lower susceptibility to, and better prognosis of blood group A of Ebola infection. It also appears though difficult to conclude due to the low numbers that persons with negative Rhesus group generally are infected less and survive more than those with a positive Rhesus group in the event of Ebola exposure, particularly if they are of blood group A.”

In the case of HIV, we have seen some interesting indications, but need more global data to ensure that weak expressions of D are clearly separated from complete gene deletions in patients.

In the case of the Black Death, it has been suggested that haemochromatosis might have been responsible for an increased survival during the plague.

” Though it is well known that the plague was responsible for the deaths of nearly half of the European population at the time, evidence also suggests that it may be responsible for the high rates of a disease called hemochromatosis; a disease of excessive iron storage, that “is the most common inherited single gene disorder in people of Northern and Western European descent. ”
” This survival advantage during the plague is thought to be responsible for the current high rates of the mutation in those with European ancestry. If true, this mutation may have been responsible for an increased survival during the plague, but at the cost of a decreased life expectancy for them later on. ”

The populations where haemochromatosis is frequent tend to also have high percentages of Rh(D) negatives.

This is just some random information I feel appropriate at this time due to keyword searches landing here. There is much more detail on this blog and you can use the search function to find it.

Don’t assume resistance or immunity based on studies of other viruses. Don’t assume resistance or immunity to any virus regardless of study.

In some cases, it has been possible to resist viruses enough to where those infected carry a low viral load and recover fast, yet while carrying it, infecting others who wind up dying. Be responsible.