Epidemiological studies have shown that individuals with different ABO(H) (histo-blood group) phenotypes are infected with NoV strains in a genotype-specific manner.[80][81] GII.4 includes global epidemic strains and binds to more histo-blood group antigens than other genogroups.[80] FUT2 fucosyl-transferase transfers a fucose sugar to the end of the ABO(H) precursor in gastrointestinal cells and saliva glands. The ABH-antigen produced is thought to act as a receptor for human norovirus: A non-functional fucosyltransferase FUT2 provides high protection from the most common norovirus strain, GII.4.[82]
Nothing yet on rh negative blood.